Varda Shoshan-Barmatz

Emailvardasbbgu.acil

h-index

Research activity per year

Fingerprint

Dive into the research topics where Varda Shoshan-Barmatz is active. These topic labels come from the works of this person. Together they form a unique fingerprint.

1 Similar Profiles

174 Article
38 Patent
17 Review article
11 Comment/debate
More

Erratum: Survivin/BIRC5-derived peptide disrupts survivin dimerization and cell division and induces multifaceted anti-cancer effects (Molecular Therapy: Oncology (2026) 34(1), (S2950329925001924), (10.1016/j.omton.2025.201123))
Santhanam, M., Babu, V., Shteinfer-Kuzmine, A., Pandey, S. K., Gheber, L., Raday, G. & Shoshan-Barmatz, V., 19 Mar 2026, In: Molecular Therapy Oncology. 34, 1, 201142.
Research output: Contribution to journal › Comment/debate

Open Access
MAVS as a Key Regulator of Tumor Proliferation, Survival, the Tumor Microenvironment, and Immunity
Trishna, S., Shteinfer-Kuzmine, A., Chalifa-Caspi, V. & Shoshan-Barmatz, V., 1 Apr 2026, In: Biomolecules. 16, 4, 501.
Research output: Contribution to journal › Article › peer-review

Open Access
p53 Interacts with VDAC1, Modulating Its Expression Level and Oligomeric State to Activate Apoptosis
Gigi, E., Karunanithi Nivedita, A., Ben-Hail, D., Santhanam, M., Shteinfer-Kuzmine, A. & Shoshan-Barmatz, V., 1 Jan 2026, In: Biomolecules. 16, 1, 141.
Research output: Contribution to journal › Article › peer-review

Open Access
Signaling pathways regulating VDAC1 overexpression associated with apoptosis, pyroptosis, and ferroptosis
Karunanithi Nivedita, A., Santhanam, M., Babu, V., Shteinfer-Kuzmine, A., Garcia Venzor, A., Toiber, D. & Shoshan-Barmatz, V., 1 Dec 2026, In: Cell Communication and Signaling. 24, 1, 120.
Research output: Contribution to journal › Article › peer-review

Open Access
Survivin/BIRC5-derived peptide disrupts survivin dimerization and cell division and induces multifaceted anti-cancer effects
Santhanam, M., Babu, V., Shteinfer-Kuzmine, A., Pandey, S. K., Gheber, L., Raday, G. & Shoshan-Barmatz, V., 19 Mar 2026, In: Molecular Therapy Oncology. 34, 1, 201123.
Research output: Contribution to journal › Article › peer-review

Open Access
2 Scopus citations
Survivin/BIRC5-derived peptide disrupts survivin dimerization and cell division and induces multifaceted anti-cancer effects
Santhanam, M., Babu, V., Shteinfer-Kuzmine, A., Pandey, S. K., Gheber, L., Raday, G. & Shoshan-Barmatz, V., 19 Mar 2026, In: Molecular Therapy Oncology. 34, 1, 201142.
Research output: Contribution to journal › Comment/debate

Open Access
Targeting VDAC1 to protect against mitochondria-linked cell death pathways: apoptosis, pyroptosis, ferroptosis, and associated diseases
Shteinfer-Kuzmine, A., Nivedita, A. K., Santhanam, M., Trishna, S., Swerdlow, R. W., Pan, J. & Shoshan-Barmatz, V., 1 Apr 2026, In: Apoptosis. 31, 4, 122.
Research output: Contribution to journal › Article › peer-review

Open Access
Annexin A5 controls VDAC1-dependent mitochondrial Ca²⁺ homeostasis and determines cellular susceptibility to apoptosis
Oflaz, F. E., Bondarenko, A. I., Trenker, M., Waldeck-Weiermair, M., Gottschalk, B., Bernhart, E., Koshenov, Z., Radulović, S., Rost, R., Hirtl, M., Pilic, J., Karunanithi Nivedita, A., Sagintayev, A., Leitinger, G., Brachvogel, B., Summerauer, S., Shoshan-Barmatz, V., Malli, R. & Graier, W. F., 16 Jun 2025, In: EMBO Journal. 44, 12, p. 3413-3447 35 p.
Research output: Contribution to journal › Article › peer-review

Open Access
8 Scopus citations
Correction to: Elevated serum mtDNA in COVID-19 patients is linked to SARS-CoV-2 envelope protein targeting mitochondrial VDAC1, inducing apoptosis and mtDNA release (Apoptosis, (2024), 29, 11-12, (2025-2046), 10.1007/s10495-024-02025-5)
Shteinfer‑Kuzmine, A., Verma, A., Bornshten, R., Chetrit, E. B., Ben‑Ya’acov, A., Pahima, H., Rubin, E., Mograbi, Y., Shteyer, E. & Shoshan‑Barmatz, V., 1 Jun 2025, In: Apoptosis. 30, 5, p. 1095 1 p.
Research output: Contribution to journal › Comment/debate

Open Access
Etoposide-induced cancer cell death: roles of mitochondrial VDAC1 and calpain, and resistance mechanisms
Karunanithi Nivedita, A. & Shoshan-Barmatz, V., 1 Jun 2025, In: Molecular Oncology. 19, 6, p. 1855-1875 21 p.
Research output: Contribution to journal › Article › peer-review

Open Access
4 Scopus citations

Ben-Gurion University of the Negev Researchers Illuminate Research in Cancer (MAVS as a Key Regulator of Tumor Proliferation, Survival, the Tumor Microenvironment, and Immunity)
Shoshan-Barmatz, V. & Chalifa-Caspi, V.
8/05/26
1 item of Media coverage
Press/Media
Ben-Gurion University of the Negev Researchers Add New Study Findings to Research in Apoptosis (p53 Interacts with VDAC1, Modulating Its Expression Level and Oligomeric State to Activate Apoptosis)
Shoshan-Barmatz, V.
10/02/26
1 item of Media coverage
Press/Media
New Findings from Ben-Gurion University of the Negev in the Area of Cancer Reported (Smac/diablo Controls Proliferation of Cancer Cells By Regulating Phosphatidylethanolamine Synthesis)
Shoshan-Barmatz, V. & Zalk, R.
19/12/22
1 item of Media coverage
Press/Media
New Apoptosis Findings from Ben-Gurion University of the Negev Described (The Multicellular Effects of VDAC1 N-Terminal-Derived Peptide)
Shoshan-Barmatz, V.
7/11/22
1 item of Media coverage
Press/Media

3 Active
6 Finished

Mitochondria
Gross, A. (PI), Shoshan-Barmatz, V. (CoPI), Pines, O. O. (CoPI), Azem, A. (CoPI) & Shirihai, O. (CoPI)
Israel Science Foundation
1/01/17 → …
Project: Research
Usage of INCPM platform on ISF research grant No.307/13 - Delineating the mechanism and functional role of the mitochondrial protein VDAC1 in apoptosis: a study of structure-function
Shoshan-Barmatz, V. (PI)
Israel Science Foundation
1/01/15 → …
Project: Research
A Novel Ryanodine Receptor in Retina: Function & Control Mechanisms
Shoshan-Barmatz, V. (PI) & Vardi, N. (CoPI)
United States-Israel Binational Science Foundation (BSF)
1/01/01 → …
Project: Research
VDAC1 overexpression and oligomerization lie at the intersection of programed cell death and disease
Shoshan-Barmatz, V. (PI)
Israel Science Foundation
1/01/23 → 31/12/25
Project: Research
VDAC1 expression levels and sub-cellular localization under pathological and apoptosis conditions: Possible control mechanisms
Shoshan-Barmatz, V. (PI)
Israel Science Foundation
1/01/19 → 31/12/22
Project: Research
VDAC1 expression levels and sub-cellular localization under pathological and apoptosis conditions: Possible control mechanisms
Shoshan-Barmatz, V. (PI)
Israel Science Foundation
1/01/19 → 31/12/22
Project: Research
Delineating the mechanism and functional role of the mitochondrial protein VDAC1 in apoptosis: a study of structure-function
Shoshan-Barmatz, V. (PI)
Israel Science Foundation
1/01/13 → 1/01/18
Project: Research
Uncovering the role of the mitochondrial protein VDAC1 in the regulation of apoptotic cell death
Shoshan-Barmatz, V. (PI)
Israel Science Foundation
1/01/09 → 31/12/12
Project: Research
Mitochondrial VDAC,the Gateway to Cell Life and Death -Structure-Function Relationship
Shoshan-Barmatz, V. (PI)
Israel Science Foundation
1/01/05 → 31/12/08
Project: Research

Varda Shoshan-Barmatz

Personal profile

Fingerprint

Collaborations and top research areas from the last five years

Dive into details

Erratum: Survivin/BIRC5-derived peptide disrupts survivin dimerization and cell division and induces multifaceted anti-cancer effects (Molecular Therapy: Oncology (2026) 34(1), (S2950329925001924), (10.1016/j.omton.2025.201123))

MAVS as a Key Regulator of Tumor Proliferation, Survival, the Tumor Microenvironment, and Immunity

p53 Interacts with VDAC1, Modulating Its Expression Level and Oligomeric State to Activate Apoptosis

Signaling pathways regulating VDAC1 overexpression associated with apoptosis, pyroptosis, and ferroptosis

Survivin/BIRC5-derived peptide disrupts survivin dimerization and cell division and induces multifaceted anti-cancer effects

Survivin/BIRC5-derived peptide disrupts survivin dimerization and cell division and induces multifaceted anti-cancer effects

Targeting VDAC1 to protect against mitochondria-linked cell death pathways: apoptosis, pyroptosis, ferroptosis, and associated diseases

Annexin A5 controls VDAC1-dependent mitochondrial Ca²⁺ homeostasis and determines cellular susceptibility to apoptosis

Correction to: Elevated serum mtDNA in COVID-19 patients is linked to SARS-CoV-2 envelope protein targeting mitochondrial VDAC1, inducing apoptosis and mtDNA release (Apoptosis, (2024), 29, 11-12, (2025-2046), 10.1007/s10495-024-02025-5)

Etoposide-induced cancer cell death: roles of mitochondrial VDAC1 and calpain, and resistance mechanisms

Ben-Gurion University of the Negev Researchers Illuminate Research in Cancer (MAVS as a Key Regulator of Tumor Proliferation, Survival, the Tumor Microenvironment, and Immunity)

Ben-Gurion University of the Negev Researchers Add New Study Findings to Research in Apoptosis (p53 Interacts with VDAC1, Modulating Its Expression Level and Oligomeric State to Activate Apoptosis)

New Findings from Ben-Gurion University of the Negev in the Area of Cancer Reported (Smac/diablo Controls Proliferation of Cancer Cells By Regulating Phosphatidylethanolamine Synthesis)

New Apoptosis Findings from Ben-Gurion University of the Negev Described (The Multicellular Effects of VDAC1 N-Terminal-Derived Peptide)

Mitochondria

Usage of INCPM platform on ISF research grant No.307/13 - Delineating the mechanism and functional role of the mitochondrial protein VDAC1 in apoptosis: a study of structure-function

A Novel Ryanodine Receptor in Retina: Function & Control Mechanisms

VDAC1 overexpression and oligomerization lie at the intersection of programed cell death and disease

VDAC1 expression levels and sub-cellular localization under pathological and apoptosis conditions: Possible control mechanisms

VDAC1 expression levels and sub-cellular localization under pathological and apoptosis conditions: Possible control mechanisms

Delineating the mechanism and functional role of the mitochondrial protein VDAC1 in apoptosis: a study of structure-function

Uncovering the role of the mitochondrial protein VDAC1 in the regulation of apoptotic cell death

Mitochondrial VDAC,the Gateway to Cell Life and Death -Structure-Function Relationship

Varda Shoshan-Barmatz

Personal profile

Fingerprint

Collaborations and top research areas from the last five years

Research output

Press/Media

Projects