Abstract
The authors regret to inform that there were unknowing errors in some figures. The corrected images are given below. The scientific conclusion of the original paper remain unchanged. Fig. 3 (h) Dark control and UVB control have been changed in CPDs formation. [Figure presented] Fig. 3 (h) BP (10 and 25 μg/mL) induced formation of cyclobutane pyrimidine dimers in HaCaT cells under UVB (1.62 J/cm2) irradiation. Control cells showed no CPDs formation. CPDs formation confirms the genotoxic potential of BP. Fig. 6 (b) UV+BP (5 μg/mL) has been changed in AnnexinV/PI trranslocation study. [Figure presented] Fig. 6 (b) BP (5 and 10 μg/mL) induced apoptosis in HaCaT cells through annexin V/FITC conjugate and propidium iodide under UVA (2.7 J/cm2) irradiation. Control cells showed no fluorescence under confocal microscope, thus imaged under differential interference contrast (DIC). Cells treated with BP (5 μg/mL) followed by UVA (2.7 J/cm2) showed decrease in cells number, translocation of PS to outer leaflet of cell membrane, shown with green fluorescence of FITC, but membrane is not too compromised to allow the PI to stain the nucleus, represents population of early apoptotic cells. Cells treated with BP (10 μg/mL) followed by UVA (2.7 J/cm2) exposure showed red stain of PI, apart from PS translocation, represents late apoptotic population. Fig. 5 (d) In JC-1 staining Dark control and Dark+ 25 μg/mL have been changed. (e) In acridine orange staining Dark+10 μg/mL has been changed. [Figure presented] Fig. 5 (d) Effect of BP (25 μg/mL) upon changes of mitochondrial membrane potential in HaCaT cells, under UVA (2.7 J/cm2) exposure through JC-1 staining. Red fluorescence of J–aggregates represents cells with high mitochondrial membrane potential as well as green fluorescence of JC-1 monomers indicates cells with low mitochondrial membrane potential, viewed under fluorescent microscope. (e) Effect of BP (10 μg/mL) on lysosomal membrane integrity of HaCaT cells through acridine orange staining under UVA (2.7 J/cm2) irradiation. Red fluorescence represents intact lysosomes as seen in control cells and orange fluorescence represents destabilized lysosomes, under fluorescence microscope. Fig. 7 (a) Western blot of pro caspase 3 and cleaved caspase 3 (c) Densitometric graph of pro caspase 3 and (d) cleaved caspase 3. [Figure presented] Fig. 7 (a) Representative Western blot image of pro caspase 3 and cleaved caspase 3. Relative densitometry analysis of pro caspase 3 (c) and cleaved caspase 3 (d). The authors would like to apologise for any inconvenience caused.
| Original language | English |
|---|---|
| Pages (from-to) | 194-195 |
| Number of pages | 2 |
| Journal | Toxicology Letters |
| Volume | 314 |
| DOIs |
|
| State | Published - 10 Oct 2019 |
| Externally published | Yes |
ASJC Scopus subject areas
- Toxicology
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Dive into the research topics of 'Corrigendum to “Role of type I and type II radicals in DNA damage and activation of Caspase 3 via mitochondrial pathway induced by photosensitized benzophenone” [Toxicol. Lett. 235 (2015) 84–95](S0378427415001113)(10.1016/j.toxlet.2015.03.008)'. Together they form a unique fingerprint.Cite this
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