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How does mTOR sense glucose starvation? AMPK is the usual suspect
Gabriel Leprivier,
Barak Rotblat
Department of Life Sciences
Research output
:
Contribution to journal
›
Article
›
peer-review
57
Scopus citations
Overview
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Dive into the research topics of 'How does mTOR sense glucose starvation? AMPK is the usual suspect'. Together they form a unique fingerprint.
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Keyphrases
Mammalian Target of Rapamycin (mTOR)
100%
Glucose Starvation
100%
AMP-activated Protein Kinase
100%
Glucose Concentration
25%
Glucose Level
25%
Glucose Availability
25%
Anabolic Effect
25%
Cellular Processes
12%
Signaling Pathway
12%
Antioxidant
12%
Biological Function
12%
Cellular Metabolism
12%
Potential Therapeutic Strategies
12%
Lack of Response
12%
Autophagic
12%
Regulation Mode
12%
Cellular Level
12%
Response Change
12%
Protein Complex
12%
Key Sensor
12%
Energy Sensor
12%
MTOR Complex 2 (mTORC2)
12%
Biological Life
12%
Mechanistic Target of Rapamycin Kinase
12%
Biochemistry, Genetics and Molecular Biology
Mechanistic Target of Rapamycin
100%
AMP-activated Protein Kinase
100%
Glucose Level
66%
Enzyme
33%
Adenosine Triphosphate
33%
Signal Transduction
33%
Biological Functions
33%
Cell Metabolism
33%
Protein Complexes
33%
Medicine and Dentistry
Mechanistic Target of Rapamycin
100%
Hydroxymethylglutaryl Coenzyme A Reductase Kinase
100%
Disease
33%
Adenosine Triphosphate
33%
Malignant Neoplasm
33%
Antioxidant
33%
Signal Transduction
33%
Biological Functions
33%
Cell Metabolism
33%
Pharmacology, Toxicology and Pharmaceutical Science
Mammalian Target of Rapamycin
100%
Hydroxymethylglutaryl Coenzyme A Reductase Kinase
100%
Disease
33%
Malignant Neoplasm
33%
Antioxidant
33%
Cellular Processes
33%
Adenosine Triphosphate
33%
Neuroscience
Mammalian Target of Rapamycin
100%
AMP-activated Protein Kinase
100%
Metabolic Pathway
33%
Cell Signaling
33%
Adenosine Triphosphate
33%
Antioxidant
33%
Protein Complexes
33%