Inflammation and accelerated atherosclerosis: Basic mechanisms

Andrea Doria; Yaniv Sherer; Pier L. Meroni; Yehuda Shoenfeld

doi:10.1016/j.rdc.2005.01.006

Inflammation and accelerated atherosclerosis: Basic mechanisms

Andrea Doria
, Yaniv Sherer
, Pier L. Meroni
, Yehuda Shoenfeld

Research output: Contribution to journal › Review article › peer-review

70 Scopus citations

Abstract

The studies described in this article support a role for immunologic-inflammatory mechanisms in the pathogenesis of atherosclerosis. This immunologic-inflammatory state is evident in many autoimmune diseases, but also in the general population lacking an overt autoimmune disease. The ability to immunomodulate atherosclerosis (currently only experimental) should lead to future research into the mechanisms and treatment of atherosclerosis, the leading cause of death in the Western world.

Original language	English
Pages (from-to)	355-362
Number of pages	8
Journal	Rheumatic Disease Clinics of North America
Volume	31
Issue number	2
DOIs	https://doi.org/10.1016/j.rdc.2005.01.006
State	Published - 1 May 2005
Externally published	Yes

UN SDGs

This output contributes to the following UN Sustainable Development Goals (SDGs)

SDG 3 Good Health and Well-being

ASJC Scopus subject areas

Rheumatology

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10.1016/j.rdc.2005.01.006

Cite this

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title = "Inflammation and accelerated atherosclerosis: Basic mechanisms",

abstract = "The studies described in this article support a role for immunologic-inflammatory mechanisms in the pathogenesis of atherosclerosis. This immunologic-inflammatory state is evident in many autoimmune diseases, but also in the general population lacking an overt autoimmune disease. The ability to immunomodulate atherosclerosis (currently only experimental) should lead to future research into the mechanisms and treatment of atherosclerosis, the leading cause of death in the Western world.",

author = "Andrea Doria and Yaniv Sherer and Meroni, \{Pier L.\} and Yehuda Shoenfeld",

year = "2005",

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T1 - Inflammation and accelerated atherosclerosis

T2 - Basic mechanisms

AU - Doria, Andrea

AU - Sherer, Yaniv

AU - Meroni, Pier L.

AU - Shoenfeld, Yehuda

PY - 2005/5/1

Y1 - 2005/5/1

N2 - The studies described in this article support a role for immunologic-inflammatory mechanisms in the pathogenesis of atherosclerosis. This immunologic-inflammatory state is evident in many autoimmune diseases, but also in the general population lacking an overt autoimmune disease. The ability to immunomodulate atherosclerosis (currently only experimental) should lead to future research into the mechanisms and treatment of atherosclerosis, the leading cause of death in the Western world.

AB - The studies described in this article support a role for immunologic-inflammatory mechanisms in the pathogenesis of atherosclerosis. This immunologic-inflammatory state is evident in many autoimmune diseases, but also in the general population lacking an overt autoimmune disease. The ability to immunomodulate atherosclerosis (currently only experimental) should lead to future research into the mechanisms and treatment of atherosclerosis, the leading cause of death in the Western world.

UR - https://www.scopus.com/pages/publications/19544366261

U2 - 10.1016/j.rdc.2005.01.006

DO - 10.1016/j.rdc.2005.01.006

M3 - Review article

C2 - 15922150

AN - SCOPUS:19544366261

SN - 0889-857X

VL - 31

SP - 355

EP - 362

JO - Rheumatic Disease Clinics of North America

JF - Rheumatic Disease Clinics of North America

IS - 2

ER -

Inflammation and accelerated atherosclerosis: Basic mechanisms

Abstract

UN SDGs

ASJC Scopus subject areas

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