Lack of effect of beta-2 adrenergic stimulation on human prolactin

Jacob Bannet; Bernard Lerer; Richard P. Ebstein; Menashe Ben-David; Robert H. Belmaker

doi:10.1016/0306-4530(81)90010-X

Lack of effect of beta-2 adrenergic stimulation on human prolactin

Jacob Bannet, Bernard Lerer, Richard P. Ebstein, Menashe Ben-David, Robert H. Belmaker

Research output: Contribution to journal › Article › peer-review

1 Scopus citations

Abstract

(1) Eleven depressive patients and one normal subject received 0.25 mg of salbutamol intravenously as part of a study of the antidepressant properties of this beta-2 adrenergic agonist. (2) No change whatsoever in the serum prolactin levels was observed after the salbutamol injection, indicating lack of involvement of beta-2 adrenergic receptor activation in the control of prolactin secretion.

Original language	English
Pages (from-to)	177-179
Number of pages	3
Journal	Psychoneuroendocrinology
Volume	6
Issue number	2
DOIs	https://doi.org/10.1016/0306-4530(81)90010-X
State	Published - 1 Jan 1981
Externally published	Yes

ASJC Scopus subject areas

Endocrinology, Diabetes and Metabolism
Endocrinology
Endocrine and Autonomic Systems
Psychiatry and Mental health
Biological Psychiatry

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10.1016/0306-4530(81)90010-X

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title = "Lack of effect of beta-2 adrenergic stimulation on human prolactin",

abstract = "(1) Eleven depressive patients and one normal subject received 0.25 mg of salbutamol intravenously as part of a study of the antidepressant properties of this beta-2 adrenergic agonist. (2) No change whatsoever in the serum prolactin levels was observed after the salbutamol injection, indicating lack of involvement of beta-2 adrenergic receptor activation in the control of prolactin secretion.",

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AU - Lerer, Bernard

AU - Ebstein, Richard P.

AU - Ben-David, Menashe

AU - Belmaker, Robert H.

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N2 - (1) Eleven depressive patients and one normal subject received 0.25 mg of salbutamol intravenously as part of a study of the antidepressant properties of this beta-2 adrenergic agonist. (2) No change whatsoever in the serum prolactin levels was observed after the salbutamol injection, indicating lack of involvement of beta-2 adrenergic receptor activation in the control of prolactin secretion.

AB - (1) Eleven depressive patients and one normal subject received 0.25 mg of salbutamol intravenously as part of a study of the antidepressant properties of this beta-2 adrenergic agonist. (2) No change whatsoever in the serum prolactin levels was observed after the salbutamol injection, indicating lack of involvement of beta-2 adrenergic receptor activation in the control of prolactin secretion.

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Lack of effect of beta-2 adrenergic stimulation on human prolactin

Abstract

ASJC Scopus subject areas

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