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MiR-608 overexpression in idiopathic pulmonary fibrosis (IPF)
Gali Epstein Shochet
, Lilach Israeli-Shani
, Isabelle Kains
, Ori Wand
, David Shitrit
Research output
:
Contribution to journal
›
Article
›
peer-review
3
Scopus citations
Overview
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Dive into the research topics of 'MiR-608 overexpression in idiopathic pulmonary fibrosis (IPF)'. Together they form a unique fingerprint.
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Keyphrases
Overexpression
100%
Idiopathic Pulmonary Fibrosis
100%
MiR-608
100%
MicroRNA
57%
Interleukin-6
42%
Cdc42
42%
Patient Samples
28%
Israeli
14%
Healthy Controls
14%
Lung
14%
Healthy Individuals
14%
Quantitative PCR
14%
Acetylcholinesterase
14%
Scar
14%
Progressive Disease
14%
Single nucleotide Polymorphism
14%
Fibrotic Diseases
14%
Formalin-fixed Paraffin-embedded Tissue
14%
Comparisons with a Control
14%
Chronic Progressive
14%
Primate-specific
14%
Whole Blood Sample
14%
Progressive Fibrosing Interstitial Lung Disease (PF-ILD)
14%
Human-specific
14%
Allele Polymorphism
14%
Usual Interstitial Pneumonia Pattern
14%
Biochemistry, Genetics and Molecular Biology
Allele
100%
MicroRNA
100%
Animal Model
25%
RNA
25%
Prevalence
25%
Acetylcholinesterase
25%
Real-Time Polymerase Chain Reaction
25%
Single-Nucleotide Polymorphism
25%
Tissue Section
25%
Scar Formation
25%
Pharmacology, Toxicology and Pharmaceutical Science
Fibrosing Alveolitis
100%
Disease
57%
microRNA
57%
Acetylcholinesterase
14%
Scar Formation
14%
Prevalence
14%
Interstitial Lung Disease
14%
Formaldehyde
14%
Interstitial Pneumonia
14%
Immunology and Microbiology
Allele
100%
Scar Formation
25%
Prevalence
25%
Single Nucleotide Polymorphism
25%
Real-Time Polymerase Chain Reaction
25%
Tissue Section
25%
Neuroscience
Pulmonary Fibrosis
100%
MicroRNA
57%
Real-Time Polymerase Chain Reaction
14%
Acetylcholinesterase
14%
Single-Nucleotide Polymorphism
14%