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Molecular mechanisms of cellular transformation by HTLV-1 Tax
Ralph Grassmann
, Mordechai Aboud
, Kuan Teh Jeang
The Shraga Segal Department of Microbiology, Immunology and Genetics
Research output
:
Contribution to journal
›
Article
›
peer-review
361
Scopus citations
Overview
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Dive into the research topics of 'Molecular mechanisms of cellular transformation by HTLV-1 Tax'. Together they form a unique fingerprint.
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Keyphrases
Protein Expression
100%
Molecular Mechanism
100%
T Cells
100%
Cell Growth
100%
Viral Replication
100%
NF-B
100%
Fibroblasts
100%
Tumor Suppressor
100%
Rodents
100%
Signaling Cascades
100%
Cell Surface Receptor
100%
Interleukin-15 (IL-15)
100%
Soluble interleukin-2 Receptor (sIL-2R)
100%
Phosphoinositide 3-kinase (PI3K)
100%
Malignant Transformation
100%
Interleukin-13 (IL-13)
100%
Cell Transformation
100%
Tax Protein
100%
Adult T-cell Leukemia
100%
Direct Binding
100%
Structural Damage
100%
Cyclin-dependent Kinase
100%
JAK-STAT
100%
Cytokine Receptor
100%
HTLV-1 Tax
100%
Mutator Phenotype
100%
Missegregation
100%
OX40
100%
Costimulatory
100%
Biochemistry, Genetics and Molecular Biology
T Cell
100%
Human T-Lymphotropic Virus 1
100%
Cell Transformation
100%
Molecular Mechanism
100%
Protein Expression
50%
Virus Replication
50%
Cytokine
50%
CREB
50%
Cell Growth
50%
P53
50%
Carcinogenesis
50%
Promoter Region
50%
Tumor Suppressor Protein
50%
Malignant Transformation
50%
Interleukin 13
50%
Cytokine Receptor
50%
Cyclin-Dependent Kinase
50%
Interleukin 15
50%
Human T Cell Leukemia Virus
50%
OX40L
50%
Fibroblast
50%
Immunology and Microbiology
Human T-Lymphotropic Virus 1
100%
T Cell
100%
Cell Transformation
100%
Cell Growth
50%
Promoter Region
50%
Cytokine
50%
Protein Expression
50%
P53
50%
Viral Replication
50%
Interleukin 15
50%
Cycline
50%
Interleukin 13
50%
Cytokine Receptor
50%
Human T Cell Leukemia Virus
50%
OX40L
50%
CD134 Antigen
50%
Fibroblast
50%
Phosphotransferase
50%
Kinase
50%