Overexpression of the mitochondrial anti-viral signaling protein, MAVS, in cancers is associated with cell survival and inflammation

Keyphrases

• Antiviral Immunity 10%
• Binding Site 10%
• Bladder Cancer 10%
• Bound Form 10%
• Cancer Cell Lines 10%
• Cancer Cell Proliferation 10%
• Cancer Therapy 10%
• Cell Inflammation 100%
• Cell Proliferation 10%
• Cell Survival 100%
• Cervical Cancer 10%
• Conductance 10%
• Host Defense 10%
• In Cancer 100%
• Inflammasome 10%
• Inflammation 10%
• Interferon Regulatory Factor 10%
• Liver Cancer 10%
• Lung Cancer 10%
• Mitochondrial Antiviral Signaling Protein 100%
• Mitochondrial Proteins 10%
• New Targets 10%
• NF-B 10%
• Nuclear Translocation 10%
• Overexpression 100%
• Patient-derived Tumors 10%
• Pattern Recognition Receptors 10%
• Protein Depletion 20%
• Protein Expression 10%
• Reduced Cell 10%
• Reduced Expression 10%
• RNA Sensing 10%
• Role in Cancer 10%
• Transcriptional Regulation 10%
• Tumor Cells 10%
• Viral 10%
• Viral Infection 20%
• Voltage-dependent Anion Channel 1 (VDAC1) 20%

Biochemistry, Genetics and Molecular Biology

• Binding Site 10%
• Cancer Cell 20%
• Cell Proliferation 20%
• Cell Survival 100%
• Conductance 10%
• Host Resistance 10%
• Inflammasome 10%
• Innate Immunity 10%
• Interferon Regulatory Factor 10%
• Mitochondrial Antiviral-Signaling Protein 100%
• Mitochondrial Protein 10%
• Pattern Recognition Receptor 10%
• Protein Expression 10%
• RNA 10%
• VDAC1 20%