Protein kinase C θ deficiency increases resistance of C57BL/6J mice to Plasmodium berghei infection-induced cerebral malaria

Keyphrases

• Protein Kinase 100%
• C57BL 100%
• Cerebral Malaria 100%
• Plasmodium Berghei 100%
• Plasmodium Berghei ANKA 41%
• Experimental Cerebral Malaria 33%
• CD4+ 16%
• Neurological Symptoms 16%
• T Cells 8%
• Tumor Necrosis Factor-α 8%
• Immune Response 8%
• Endothelial Cells 8%
• Interferon-γ 8%
• Ablation 8%
• LFA-1 8%
• Acute Phase 8%
• CD8+ T 8%
• CD8+ T Cells 8%
• Cell-mediated Immune Response 8%
• Signaling Cascades 8%
• T Cell Activation 8%
• Early Onset 8%
• Immunological Synapse 8%
• Splenomegaly 8%
• Th1 Cytokines 8%
• Early Death 8%
• CD28 8%
• Sequester 8%
• Neurological Signs 8%
• T Cell Antigen Receptor 8%
• Host Response to Infection 8%
• Partial Resistance 8%
• Parasite Burden 8%
• Splenic T Cells 8%
• Lymphotoxin 8%
• Brain Microvessels 8%
• Sign Characteristic 8%

Immunology and Microbiology

• C57BL 6 Mouse 100%
• Plasmodium berghei 100%
• Mouse 83%
• Immune Response 33%
• CD4 33%
• Cytotoxic T-Cell 33%
• T Cell 16%
• Infectious Agent 16%
• Cytokine 16%
• Interferon Gamma 16%
• Lymphocyte Function-Associated Antigen 1 16%
• Adoptive Immunity 16%
• Tumor Necrosis Factor Alpha 16%
• T Cell Activation 16%
• CD28 16%
• Immunological Synapse 16%
• T Lymphocyte Antigen 16%
• Parasite Load 16%
• Microvasculature 16%
• Splenic T Cell 16%
• Lymphotoxin 16%